Turkesterone (Ajuga Turkestanica Extract) is an ecdysteroid.

Ecdysteroids are naturally occuring plant and arthropod growth-promotants. They have been shown in research to be extremely effective at building muscle and improving performance. Ecdysteroids occur naturally in foods such as quinoa, spinach, and yams. However, the amount contained in these foods is far too small to be effective.

Turkesterone has been studied for decades, and the results have been encouraging. Turkesterone has been shown to promote lipid and carbohydrate metabolism, increase protein synthesis, aid in muscle hypertrophy, and increase strength.

These results suggest that Turkesterone may act as an agonist for anabolic and adaptogenic receptors (estrogen receptor beta), while its potential lipid and carbohydrate metabolising effects may aid in fat loss and maintenance of a healthy body composition.

Critically, this product is non-suppressive, having no effect on follicle stimulating hormone production, or luteinising hormone production. This makes it safe to use for men and women.

In mitochondria, Turkesterone boosts oxidative phosphorylation which encourages cells to release more energy and more produce more adenosine triphosphate (ATP). This is critical for the contraction of muscles, boosting strength and endurance.

Protein biosynthesis is also improved by supplementing with Turkesterone. Stimulation of protein biosynthesis was due to an increase in functional activity of polyribosomes and to elevation in the synthesis of protein molecules. This is critical for building muscle tissue.

Another interesting consequence of using this supplement is increased red blood cell count. Repeated administration increased the content of erythrocytes and hemoglobin in the blood of intact rats, also causing a marked effect on red blood regeneration in hemotoxic phenylhydrazine anemia. In its capacity for simulating erythropoiesis turkesterone resembles the well-known steroidal anabolic drug nerobol.

Turkesterone also lowers stress levels by prevent involution of the thymicolymphatic system. It also prevents an increase in the weight of adrenal glands and a decrease in them of the content of ascorbic acid and cholesterol (to a much greater degree than does T-activin). In turn, turkesterone stimulates immunogenesis in animals with experimental immune deficiency developed on the background of immobilization-induced stress.